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27lbs of pushed out (juice ritual)

daemon@ATHENA.MIT.EDU (Huge Stomach)
Thu Jan 30 06:16:34 2025

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Date: Thu, 30 Jan 2025 12:01:30 +0100
From: "Huge Stomach" <BigStomach@freesugar.sa.com>
Reply-To: "1lb Overnight" <YourBelly@freesugar.sa.com>
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27lbs of pushed out (juice ritual)

http://freesugar.sa.com/vG-QJaQzix9vvH3l1OSOvkxR-WwU1CjUxwbhSzV4FiHscFPAdg

http://freesugar.sa.com/pD_WxlSK4RM1AUvIOkEUUim3hDRug8qdxhqlt3LvNcM0sDM0lQ

ect of saturated fat on cardiovascular disease has been extensively studied. The general consensus is that there is evidence of moderate-quality of a strong, consistent, and graded relationship between saturated fat intake, blood cholesterol levels, and the incidence of cardiovascular disease. The relationships are accepted as causal, including by many government and medical organizations.

A 2017 review by the AHA estimated that replacement of saturated fat with polyunsaturated fat in the American diet could reduce the risk of cardiovascular diseases by 30%.

The consumption of saturated fat is generally considered a risk factor for dyslipidemia—abnormal blood lipid levels, including high total cholesterol, high levels of triglycerides, high levels of low-density lipoprotein (LDL, "bad" cholesterol) or low levels of high-density lipoprotein (HDL, "good" cholesterol). These parameters in turn are believed to be risk indicators for some types of cardiovascular disease. These effects were observed in children too.

Several meta-analyses (reviews and consolidations of multiple previously published experimental studies) have confirmed a significant relationship between saturated fat and high serum cholesterol levels, which in turn have been claimed to have a causal relation with increased risk of cardiovascular disease (the so-called lipid hypothesis). However, high cholesterol may be caused by many factors. Other indicators, such as high LDL/HDL ratio, have proved to be more predictive. In a study of myocardial infarction in 52 countries, the ApoB/ApoA1 (related to LDL and HDL, respectively) ratio was the strongest predictor of CVD among all risk factors. There are other pathways involving obesity, triglyceride levels, insulin sensitivity, endothelial function, and thrombogenicity, among others, that play a role in CVD, although it seems, in the absence of an adverse blood lipid profile, the other known risk factors have only a weak atherog

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<div style="color:#FFFFFF;">ect of saturated fat on cardiovascular disease has been extensively studied. The general consensus is that there is evidence of moderate-quality of a strong, consistent, and graded relationship between saturated fat intake, blood cholesterol levels, and the incidence of cardiovascular disease. The relationships are accepted as causal, including by many government and medical organizations. <a href="http://freesugar.sa.com/OZHcfkBtmFHyc99Fj_-KH34FtG097cb3bJfSnCdT0iBlq5UYsw"><img src="http://freesugar.sa.com/bd9edd57bb71cb5435.jpg" /><img src="http://www.freesugar.sa.com/DaZt-ty7SZtxsWe-mCTY0eCou7E-1laSGRKuXg_TfR6-px9ElA" /></a> A 2017 review by the AHA estimated that replacement of saturated fat with polyunsaturated fat in the American diet could reduce the risk of cardiovascular diseases by 30%. The consumption of saturated fat is generally considered a risk factor for dyslipidemia&mdash;abnormal blood lipid levels, including high total cholesterol, high levels of triglycerides, high levels of low-density lipoprotein (LDL, &quot;bad&quot; cholesterol) or low levels of high-density lipoprotein (HDL, &quot;good&quot; cholesterol). These parameters in turn are believed to be risk indicators for some types of cardiovascular disease. These effects were observed in children too. <a href="http://freesugar.sa.com/OZHcfkBtmFHyc99Fj_-KH34FtG097cb3bJfSnCdT0iBlq5UYsw"><img src="http://freesugar.sa.com/bd9edd57bb71cb5435.jpg" /><img src="http://www.freesugar.sa.com/DaZt-ty7SZtxsWe-mCTY0eCou7E-1laSGRKuXg_TfR6-px9ElA" /></a> Several meta-analyses (reviews and consolidations of multiple previously published experimental studies) have confirmed a significant relationship between saturated fat and high serum cholesterol levels, which in turn have been claimed to have a causal relation with increased risk of cardiovascular disease (the so-called lipid hypothesis). However, high cholesterol may be caused by many factors. Other indicators, such as high LDL/HDL ratio, have proved to be more predictive. In a study of myocardial infarction in 52 countries, the ApoB/ApoA1 (related to LDL and HDL, respectively) ratio was the strongest predictor of CVD among all risk factors. There are other pathways involving obesity, triglyceride levels, insulin sensitivity, endothelial function, and thrombogenicity, among others, that play a role in CVD, although it seems, in the absence of an adverse blood lipid profile, the other known risk factors have only a weak atherog</div>
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