[48219] in linux-announce channel archive
Save THOUSANDS on future vehicle repair bills!
daemon@ATHENA.MIT.EDU (Vehicle Protection Info)
Fri Apr 11 09:29:24 2025
Date: Fri, 11 Apr 2025 08:28:50 -0500
From: "Vehicle Protection Info" <StopUnnecessaryCosts@neuroquite.ru.com>
Reply-To: "Vehicle Protection" <VehicleServicePlan@neuroquite.ru.com>
To: <linuxch-announce.discuss@charon.mit.edu>
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Save THOUSANDS on future vehicle repair bills!
http://neuroquite.ru.com/_Ov9kK2p6Z7yW99EiPY6Q7VCEHVB9xDGFUALy8nUDhj9uvkdeg
http://neuroquite.ru.com/PBX3kDJCVNyeHSbAxOu1FuuzkhWDsSGY3Fvc_JhUFvB9YrAc0g
phism can be controlled by alleles at a single locus (e.g. human ABO blood groups), the more complex forms are controlled by supergenes consisting of several tightly linked genes on a single chromosome. Batesian mimicry in butterflies and heterostyly in angiosperms are good examples. There is a long-standing debate as to how this situation could have arisen, and the question is not yet resolved.
Whereas a gene family (several tightly linked genes performing similar or identical functions) arises by duplication of a single original gene, this is usually not the case with supergenes. In a supergene some of the constituent genes have quite distinct functions, so they must have come together under selection. This process might involve suppression of crossing-over, translocation of chromosome fragments and possibly occasional cistron duplication. That crossing-over can be suppressed by selection has been known for many years.
Debate has centered round the question of whether the component genes in a super-gene could have started off on separate chromosomes, with subsequent reorganization, or if it is necessary for them to start on the same chromosome. Originally, it was held that chromosome rearrangement would play an important role. This explanation was accepted by E. B. Ford and incorporated into his accounts of ecological genetics.:?ch. 6?:?17–25?
However, many believe it more likely that the genes start on the same chromosome. They argue that supergenes arose in situ. This is known as Turner's sieve hypothesis. John Maynard Smith agreed with this view in his authoritative textbook, but the question is still not defin
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<div style="color:#FFFFFF;font-size:8px;">phism can be controlled by alleles at a single locus (e.g. human ABO blood groups), the more complex forms are controlled by supergenes consisting of several tightly linked genes on a single chromosome. Batesian mimicry in butterflies and heterostyly in angiosperms are good examples. There is a long-standing debate as to how this situation could have arisen, and the question is not yet resolved. Whereas a gene family (several tightly linked genes performing similar or identical functions) arises by duplication of a single original gene, this is usually not the case with supergenes. In a supergene some of the constituent genes have quite distinct functions, so they must have come together under selection. This process might involve suppression of crossing-over, translocation of chromosome fragments and possibly occasional cistron duplication. That crossing-over can be suppressed by selection has been known for many years. Debate has centered round the question of whether the component genes in a super-gene could have started off on separate chromosomes, with subsequent reorganization, or if it is necessary for them to start on the same chromosome. Originally, it was held that chromosome rearrangement would play an important role. This explanation was accepted by E. B. Ford and incorporated into his accounts of ecological genetics.:?ch. 6?:?17–25? However, many believe it more likely that the genes start on the same chromosome. They argue that supergenes arose in situ. This is known as Turner's sieve hypothesis. John Maynard Smith agreed with this view in his authoritative textbook, but the question is still not defin</div>
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